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MicroRNA-135b as therapeutic target in cancers. Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. Chemokine receptors on the defensive the surprising role of CXCR4 in brown adipose tissue. Regulation of ryanodine receptor-mediated calcium signaling by presenilins. Ca2 dyshomeostasis is a critical causative mechanism underlying the functional impairment of the central nervous system seen in ‘healthy’ aging [1], specifically in normal brain aging processes in the absence of disease-causing mu...

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MicroRNA-135b as therapeutic target in cancers. Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. Chemokine receptors on the defensive the surprising role of CXCR4 in brown adipose tissue. Regulation of ryanodine receptor-mediated calcium signaling by presenilins. Ca2 dyshomeostasis is a critical causative mechanism underlying the functional impairment of the central nervous system seen in ‘healthy’ aging [1], specifically in normal brain aging processes in the absence of disease-causing mu...
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Smart Science Technology | journals.smartscitech.com Reviews

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MicroRNA-135b as therapeutic target in cancers. Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. Chemokine receptors on the defensive the surprising role of CXCR4 in brown adipose tissue. Regulation of ryanodine receptor-mediated calcium signaling by presenilins. Ca2 dyshomeostasis is a critical causative mechanism underlying the functional impairment of the central nervous system seen in ‘healthy’ aging [1], specifically in normal brain aging processes in the absence of disease-causing mu...

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MicroRNA-135b as therapeutic target in cancers. Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. Chemokine receptors on the defensive the surprising role of CXCR4 in brown adipose tissue. Regulation of ryanodine receptor-mediated calcium signaling by presenilins. Ca2 dyshomeostasis is a critical causative mechanism underlying the functional impairment of the central nervous system seen in ‘healthy’ aging [1], specifically in normal brain aging processes in the absence of disease-causing mu...

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Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. MiR-126 and miR-223 as biomarkers of vascular damage in the course of Chronic Kidney Disease. Ets-1 activation, when tumors crosstalk with their microenvironment. Cancer Cell and Microenvironment. Estrogen receptor-α signaling and localization regulates autophagy and unfolded protein response activation in ER breast cancer. Receptors and Clinical Investigation. Receptors and Clinical Investigation. Receptors and Clinical Investigation. Postt...

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Inflammation and Cell Signaling. Inflammation and Cell Signaling (ISSN 2330-7803) is an open access, peer-reviewed, online journal publishing articles of the highest quality pertaining to the fields of b. Receptors and Clinical Investigation. Receptors and Clinical Investigation (ISSN 2330-0566) is an open access, peer-reviewed, online journal publishing articles of the highest quality pertaining to the fields o. Cancer Cell and Microenvironment. Molecular and Cellular Epilepsy. Immunoendocrinology (ISSN...

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Smart Science and Technology LLC. Smart Science and Technology ( www.smartscitech.com. Is a Houston publisher serving international science and technology communities with open access peer-reviewed journals covering frontier disciplines. All of the Journals published by Smart Science and Technology maintain the highest standards, with internationally-recognized editors and reviewers serving those journals. Names and addresses of the owners of the publishing company. The owner: Dr. Xinjie Tan. Research As...

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MicroRNA-135b as therapeutic target in cancers. Rescue of DNA damage-stalled RNA Pol II: histone H2B in action. Chemokine receptors on the defensive the surprising role of CXCR4 in brown adipose tissue. Regulation of ryanodine receptor-mediated calcium signaling by presenilins. Ca2 dyshomeostasis is a critical causative mechanism underlying the functional impairment of the central nervous system seen in ‘healthy’ aging [1], specifically in normal brain aging processes in the absence of disease-causing mu...

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